Lkb1 maintains Treg cell lineage identity

نویسندگان

  • Di Wu
  • Yuechen Luo
  • Wei Guo
  • Qing Niu
  • Ting Xue
  • Fei Yang
  • Xiaolei Sun
  • Song Chen
  • Yuanyuan Liu
  • Jingru Liu
  • Zhina Sun
  • Chunxiao Zhao
  • Huifang Huang
  • Fang Liao
  • Zhongchao Han
  • Dongming Zhou
  • Yongguang Yang
  • Guogang Xu
  • Tao Cheng
  • Xiaoming Feng
چکیده

Regulatory T (Treg) cells are a distinct T-cell lineage characterized by sustained Foxp3 expression and potent suppressor function, but the upstream dominant factors that preserve Treg lineage-specific features are mostly unknown. Here, we show that Lkb1 maintains Treg cell lineage identity by stabilizing Foxp3 expression and enforcing suppressor function. Upon T-cell receptor (TCR) stimulation Lkb1 protein expression is upregulated in Treg cells but not in conventional T cells. Mice with Treg cell-specific deletion of Lkb1 develop a fatal early-onset autoimmune disease, with no Foxp3 expression in most Treg cells. Lkb1 stabilizes Foxp3 expression by preventing STAT4-mediated methylation of the conserved noncoding sequence 2 (CNS2) in the Foxp3 locus. Independent of maintaining Foxp3 expression, Lkb1 programs the expression of a wide spectrum of immunosuppressive genes, through mechanisms involving the augmentation of TGF-β signalling. These findings identify a critical function of Lkb1 in maintaining Treg cell lineage identity.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017